Ad Mundo Exteriore,
delentyevox, you have nearly all of the anime mp3s I've been looking for in the past few months! Thank Apple for iTunes file sharing. ^_^ I didn't even know they made iTunes for Windows until yesterday, when I went to look at the new features in Panther. (That was actually faintly depressing since I realized that my iBook was now outdated by two OS X versions.)
I actually prefer iTunes to Winamp even if you can't make skins for it. Possibly because all my music files for the past year until I got my new PC desktop had been stored on my iBook. The file information settings for most of my old files became very strange when I switched to Winamp. But now all I have to do is change the information for the new files, which is not as much of a headache. (Well, I like having all of my mp3s listed according to a consistent format! I have a whole system going here, you know.)
I served as lector for the first time at today's student Mass. (I was substituting for someone else.) Appropriately enough, while I was reading the general intersessions, a mouse appeared out of nowhere and ran across the floor, unwittingly wreaking havoc on the way. I didn't know what to do, so I just kept reading. Honestly, people have no discipline whatsoever. Later, during communion, the mouse materialized once more and ran under my chair, but I didn't even flinch. I mean, it was a tiny mouse, barely the size of my palm. (Very cute, come to think of it. Hardly worth shrieking over.)
I wasn't entirely nonproductive this weekend, but not at full capacity either. So I won't pull a useless all-nighter. However, I do think I'll stay up an extra hour to read over the chapter on recombination in prokaryotes. I finished the chapter on chromosomal mutations about an hour ago. It was beautifully detailed and so well-organized--I love a textbook that has good structure. Each type of chromosomal mutation was classified and analyzed according to its causes and its effect on phenotype. Unfortunately, I don't think I've really absorbed all the details. I gathered that gene imbalance was one of the basic principles governing the effects of different chromosomal mutations, but I didn't really follow all the reasoning behind why inversions result in lower recombinant frequency. Well, all right, so I do understand that when there are crossovers in a chromosome containing an inversion mutation, the recombinants are not viable, which means that the frequency of recombination in the surviving offspring is much lower than expected from the map distance. But what I didn't bother trying to follow was the exact mechanism by which the meiotic products become inviable. Something to do with looping, but it's pretty confusing, especially since I half-skimmed through those sections.
On the other hand, I think I understand how the pairing occurs in reciprocal translocations. The diagram at least is a lot more intuitive. The pairing of homologous chromosomes seems to be significant for analyzing how chromosomal rearrangements cause changes in meiosis (and therefore affect the fertility of the organism), but I didn't really bother thinking it through for the inversions. I should probably review this chapter sometime before the next exam. On the other hand, the professor did say that he wouldn't test us on the mechanistic details. Still, it might be worth reading again just to finish the picture, so to speak.
(Just because you only need a few fundamental structural elements to keep an organized system of knowledge standing doesn't mean that there is a niggling sense of dissatisfaction if you don't tighten all the bolts and add the supposedly more minor rafters and columns for reinforcement. The unity and cohesiveness of the completed product is much more beautiful than the functional, but all too basic, sketch you had before. Yes, it's another long, silly metaphor.)
Off to read about plasmids! ^_^ I really would completely enjoy this class if the pace was a little slower and if I didn't have to do the stupid HGDRP.
...Tari
I actually prefer iTunes to Winamp even if you can't make skins for it. Possibly because all my music files for the past year until I got my new PC desktop had been stored on my iBook. The file information settings for most of my old files became very strange when I switched to Winamp. But now all I have to do is change the information for the new files, which is not as much of a headache. (Well, I like having all of my mp3s listed according to a consistent format! I have a whole system going here, you know.)
I served as lector for the first time at today's student Mass. (I was substituting for someone else.) Appropriately enough, while I was reading the general intersessions, a mouse appeared out of nowhere and ran across the floor, unwittingly wreaking havoc on the way. I didn't know what to do, so I just kept reading. Honestly, people have no discipline whatsoever. Later, during communion, the mouse materialized once more and ran under my chair, but I didn't even flinch. I mean, it was a tiny mouse, barely the size of my palm. (Very cute, come to think of it. Hardly worth shrieking over.)
I wasn't entirely nonproductive this weekend, but not at full capacity either. So I won't pull a useless all-nighter. However, I do think I'll stay up an extra hour to read over the chapter on recombination in prokaryotes. I finished the chapter on chromosomal mutations about an hour ago. It was beautifully detailed and so well-organized--I love a textbook that has good structure. Each type of chromosomal mutation was classified and analyzed according to its causes and its effect on phenotype. Unfortunately, I don't think I've really absorbed all the details. I gathered that gene imbalance was one of the basic principles governing the effects of different chromosomal mutations, but I didn't really follow all the reasoning behind why inversions result in lower recombinant frequency. Well, all right, so I do understand that when there are crossovers in a chromosome containing an inversion mutation, the recombinants are not viable, which means that the frequency of recombination in the surviving offspring is much lower than expected from the map distance. But what I didn't bother trying to follow was the exact mechanism by which the meiotic products become inviable. Something to do with looping, but it's pretty confusing, especially since I half-skimmed through those sections.
On the other hand, I think I understand how the pairing occurs in reciprocal translocations. The diagram at least is a lot more intuitive. The pairing of homologous chromosomes seems to be significant for analyzing how chromosomal rearrangements cause changes in meiosis (and therefore affect the fertility of the organism), but I didn't really bother thinking it through for the inversions. I should probably review this chapter sometime before the next exam. On the other hand, the professor did say that he wouldn't test us on the mechanistic details. Still, it might be worth reading again just to finish the picture, so to speak.
(Just because you only need a few fundamental structural elements to keep an organized system of knowledge standing doesn't mean that there is a niggling sense of dissatisfaction if you don't tighten all the bolts and add the supposedly more minor rafters and columns for reinforcement. The unity and cohesiveness of the completed product is much more beautiful than the functional, but all too basic, sketch you had before. Yes, it's another long, silly metaphor.)
Off to read about plasmids! ^_^ I really would completely enjoy this class if the pace was a little slower and if I didn't have to do the stupid HGDRP.
...Tari
(no subject)
Date: 2003-10-27 12:14 am (UTC)(no subject)
Date: 2003-10-27 11:19 am (UTC)...Tari
(no subject)
Date: 2003-10-27 10:22 am (UTC)(no subject)
Date: 2003-10-27 11:18 am (UTC)...Tari